As of July 31, 2026
Psychiatry & Neurology
Small molecule
DSP-0038
| Origin |
in-house (Joint research with Recursion (formerly Exscientia
Ltd.))
|
| Formulation |
oral |
| Planned indication |
- Alzheimer’s disease psychosis
|
DSP-0038 is a novel compound discovered at Sumitomo Pharma using
Recursion’s AI technologies. DSP-0038 is a serotonin 5-HT2A
receptor antagonist and a serotonin 5-HT1A receptor
agonist. DSP-0038 is expected to demonstrate a great antipsychotic
effect, based on the additive effect of 5-HT2A receptor
antagonist and 5-HT1A receptor agonist. The compound
could also have broader efficacy in the treatment of behavioral and
psychological symptoms of dementia (BPSD) which include agitation,
aggression, anxiety, and depression. Furthermore, DSP-0038 has no
dopamine D2 receptor antagonistic activity, and therefore
it can be expected to show improved safety and tolerability compared
to existing antipsychotics.
DSP-0187
| Origin |
in-house |
| Formulation |
oral |
| Planned indication |
|
DSP-0187 is a selective orexin 2 receptor agonist. It is expected to
improve excessive daytime sleepiness (EDS) and cataplexy of
narcolepsy caused by orexin deficiency. DSP-0187 is also expected to
have potential in hypersomnia disorders other than narcolepsy.
DSP-3456
| Origin |
in-house |
| Formulation |
oral |
| Planned indication |
- Treatment resistant depression
|
DSP-3456 is a metabotropic glutamate receptor 2/3 negative
allosteric modulator (mGluR2/3 NAM). DSP-3456 is expected to exhibit
a ketamine-like antidepressant effect through selective activation
of the prefrontal cortex via enhanced glutamate release, while
avoiding side effects (psychotic symptoms, cognitive dysfunction).
DSP-0378
| Origin |
in-house |
| Formulation |
oral |
| Planned indications |
-
Progressive Myoclonic Epilepsy and Developmental Epileptic
Encephalopathy
|
DSP-0378 is a gamma-aminobutyric acid (GABA)A receptor
positive allosteric modulator. DSP-0378 acts on various subtypes of
GABAA receptors expressed in synaptic and extrasynaptic
regions in a manner different from common GABAA receptor
potentiators such as benzodiazepines and neurosteroids. DSP-0378 is
expected to exhibit an antiepileptic effect against a broad range of
epilepsies including Progressive Myoclonic Epilepsy and
Developmental Epileptic Encephalopathy.
DSP-2342
| Origin |
in-house (Joint research with Recursion (formerly Exscientia
Ltd.))
|
| Formulation |
oral |
| Planned indication |
|
DSP-2342 is a novel compound discovered at Sumitomo Pharma using
Recursion’s AI technologies. DSP-2342 is a serotonin 5-HT2A
and 5-HT7 receptor antagonist. DSP-2342 is expected to
demonstrate a broad antipsychotic effect in psychosis, anxiety, and
depression, based on the dual antagonism of 5-HT2A and
5-HT7 receptors. Furthermore, DSP-2342 has high
selectivity for 5-HT2A and 5-HT7
receptors, and is therefore expected to show a high level of safety
and tolerability.
DSP-0551
| Origin |
in-house |
| Formulation |
oral |
| Planned indication |
- Tremor associated with Parkinson’s disease
|
DSP-0551 is a multi-ion channel modulator identified through
phenotype-based drug discovery. It exhibits inhibitory activity
against multiple calcium and sodium ion channels that have been
implicated in tremor. Through these mechanisms, DSP-0551 is expected
to modulate abnormal neuronal firing and excessive synchronization
in neural circuits associated with tremor, thereby improving tremor
associated with Parkinson’s disease.
Regenerative medicine / cell therapy (Collaboration with RACTHERA
Co., Ltd.)
In collaboration with RACTHERA Co., Ltd., and our partners in the
industry-academia collaboration, we are advancing the research and
development of regenerative medicine / cell therapy using iPS cells
for the treatment of Parkinson's disease, RPE (retinal pigment
epithelium) tear, AMD (age-related macular degeneration), retinitis
pigmentosa, and spinal cord injury.
CT1-DAP001/DSP-1083 (Allogeneic iPS cell-derived dopaminergic neural
progenitor cells)
| Partnering |
Kyoto University CiRA, UC San Diego, and others |
| Planned indication |
|
In Japan, CT1‑DAP001/DSP‑1083 obtained manufacturing and marketing
approval under the conditional and time‑limited approval framework
in March 2026 for the indication of the improvement of motor
symptoms in patients with Parkinson’s disease who have an inadequate
response to existing pharmacological therapies, including
levodopa-containing products. The product name is AMCHEPRY® (INN:
raguneprocel).
In the U.S., an investigator‑initiated clinical study is being
conducted at UC San Diego and other institutions, while
company‑sponsored studies are also being pursued in parallel.
HLCR011 (Allogeneic iPS cell-derived retinal pigment epithelial
cells)
| Partnering |
HEALIOS K.K., Kyushu University, and others |
| Planned indication |
- Retinal pigment epithelium tear
|
DSP-3077 (Allogeneic iPS cell-derived retinal sheet)
| Partnering |
Massachusetts Eye and Ear in Boston, Massachusetts (Teaching
hospital of Harvard Medical School) and the University of
Minnesota
|
| Planned indication |
|
The U.S. Food and Drug Administration (FDA) granted Orphan Drug
Designation for the indication of retinitis pigmentosa in March
2026.
Oncology
Enzomenib
| Origin |
in-house (Joint research with Kyoto University) |
| Formulation |
oral |
| Planned indication |
|
-
Enzomenib (DSP-5336) is a small molecule inhibitor of the menin
and lysine methyltransferase 2A (KMT2A) protein interaction. Acute
myeloid leukemia with KMT2A rearrangement or nucleophosmin 1
(NPM1) mutation relies on the menin-KMT2A interaction for
upregulation of genes instrumental to leukemogenesis. Enzomenib
has been shown to have anti-cancer activity through downregulation
of these genes by inhibition of menin-KMT2A interaction in
preclinical studies. The FDA granted Orphan Drug Designation for
enzomenib for the indication of acute myeloid leukemia in June
2022 and for acute lymphoblastic leukemia in July 2026, and
granted Fast Track Designation for the treatment of relapsed or
refractory acute myeloid leukemia with KMT2A rearrangement or NPM1
mutation in June 2024. Furthermore, the MHLW granted Orphan Drug
Designation for enzomenib for the indication of relapsed or
refractory acute myeloid leukemia with KMT2A rearrangement or NPM1
mutation in September 2024.
Nuvisertib
| Origin |
in-house (former Tolero Pharmaceuticals, Inc.) |
| Formulation |
oral |
| Planned indication |
|
-
Nuvisertib (TP-3654) inhibits the inflammatory signaling pathways
through inhibition of PIM1 (proviral integration site for Moloney
murine leukemia virus 1) kinase. PIM1 kinase is frequently
overexpressed in various hematologic malignancies and solid
tumors, allowing cancer cells to evade apoptosis and promoting
tumor growth. The FDA, the MHLW, and the European Medicines Agency
(EMA) granted Orphan Drug Designation for nuvisertib for the
indication of myelofibrosis in May 2022, November 2024, and July
2025, respectively. Additionally, the FDA granted nuvisertib Fast
Track Designation in June 2025, also for the indication of
myelofibrosis.
SMP-3124
| Origin |
in-house |
| Formulation |
injection (Liposomal Nanomedicine) |
| Planned indication |
|
-
SMP-3124 is an injectable formulation of a CHK1 inhibitor
encapsulated in PEGylated liposomes. CHK1 is a serine/threonine
kinase activated by the DNA damage response, leading to cell-cycle
arrest and DNA repair. CHK1 inhibition leads cancer cells with
high replication stress to apoptosis by inducing further DNA
damage. SMP-3124 is expected to enhance the anti-tumor activity
and reduce side effects of the CHK1 inhibitor through optimized
pharmacokinetics of the compound by liposomal nanoparticle
formulation.
DSP-0390
| Origin |
in-house |
| Formulation |
oral |
| Planned indication |
|
-
DSP-0390 is an inhibitor of Emopamil Binding Protein (EBP), an
endoplasmic reticulum membrane protein involved in cholesterol
biosynthesis. EBP mediates de novo cholesterol synthesis for cell
membrane structure and signaling, enabling aberrant growth of
tumors. Inhibition of EBP causes depletion of cellular
cholesterol, which is expected to lead to anti-cancer activity.
The FDA granted Orphan Drug Designation for DSP-0390 for the
indication of brain tumor in May 2022.
Others
KSP-1007
| Origin |
in-house (Joint research with The Kitasato Institute) |
| Formulation |
injection |
| Planned indications |
-
Complicated urinary tract infections and complicated
intra-abdominal infections, hospital-acquired bacterial
pneumonia including ventilator-associated bacterial
pneumonia
|
-
KSP-1007 broadly and strongly inhibits β-lactamases,
enzymes produced by bacteria that can degrade carbapenem
antibiotics. KSP-1007 is expected to become an effective treatment
option against carbapenem-resistant bacterial infections as a
component of a combination drug with meropenem hydrate, a
carbapenem antibiotic in general use worldwide (name of Sumitomo
Pharma’s product for the Japanese market: MEROPEN®). The FDA
granted Qualified Infectious Disease Product (QIDP) status and
Fast Track Designation for KSP-1007 for the indications of
complicated urinary tract infections, complicated intra-abdominal
infections, and hospital-acquired bacterial pneumonia including
ventilator-associated bacterial pneumonia in August 2022.
fH1/DSP-0546LP
| Origin |
in-house (Joint research with the National Institutes of
Biomedical Innovation, Health and Nutrition)
|
| Formulation |
injection |
| Planned indication |
- Influenza virus prophylaxis
|
-
fH1/DSP-0546LP is a next-generation candidate vaccine formulation
composed of the post-fusion hemagglutinin antigen (fH1) that is
expected to be effective against a broad range of influenza
viruses, and TLR7 adjuvant “DSP-0546LP” that enhances the
quantity, quality, and durability of immune responses.
Conventional influenza vaccines lose effectiveness due to viral
mutations, making it necessary to select, produce, and inoculate a
vaccine to immunize against strains predicted to circulate each
year. They may also not respond well to emerging strains of
influenza.
-
The pre-clinical study of fH1/DSP-0546LP demonstrated broad cross
protection against influenza viruses antigenically different from
those used in vaccine formulations, and the significance of the
TLR7 adjuvant, DSP-0546LP. It is expected that fH1/DSP-0546LP will
improve the breadth and durability of protection against seasonal
influenza viruses and will be effective against novel and
potentially pandemic strains.